| [ Summary ] |
Intimal hyperplasia often develops in the venous walls after arteriovenous fistula placement. The venous wall may be injured by high arterial flows. Pathologically, intimal hyperplasia consists of smooth muscle cell proliferation and migration within the neointima. Intimal hyperplasia commonly develops in the graft vein anastomotic site. Although, various studies on factors such as anastomotic angle change, cuff technique application, and tapered graft placement have been investigated, it is difficult to suppress intimal hyperplasia formation. Various drugs, radiation, and far infrared rays, may provide suppression of intimal hyperplasia development in relation to venous anastomosis. |